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c57bl 6j wild type wt counterparts  (Jackson Laboratory)

 
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    Jackson Laboratory c57bl 6j wild type wt counterparts
    C57bl 6j Wild Type Wt Counterparts, supplied by Jackson Laboratory, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/wild+type+wt+c57bl+6j/6j+c57bl+mice+type+wild/pmc13273473-340-16-23
    Average 86 stars, based on 1 article reviews
    c57bl 6j wild type wt counterparts - by Bioz Stars, 2026-09
    86/100 stars

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    other:

    Article Title: cGAS-STING dependent type I IFN reduces Leptospira interrogans renal colonization in mice
    Article Snippet: Wild-type (WT) C57BL/6J (JAX stock# 000664) mice were purchased from Jackson laboratories and used as controls unless otherwise stated.

    Article Title: Protective role of complement signaling in Kawasaki disease vasculitis.
    Article Snippet: Wild-type (WT) C57BL/6J, C3-/- (#029661), C1qa-/- 396 (#031675), and Mbl-/- (#006122) mice were obtained from the Jackson Laboratory (Bar Harbor, 397 ME, USA).

    Mouse Assay:

    Article Title: Genetic inhibition of IL-12β suppresses systolic overload-induced cardiac oxidative stress, inflammation, and heart failure development
    Article Snippet: .. Mice and Experimental Protocols: Male and female IL-12β knockout (Jackson Laboratory, Strain #002693) and wild-type (WT) C57BL/6J (Jackson Laboratory, Strain #000664) mice were subjected to sham or transverse aortic constriction (TAC) surgery, a procedure used to replicate clinical conditions such as hypertension and aortic stenosis. ..

    Article Title: Genetic inhibition of IL-12β suppresses systolic overload-induced cardiac oxidative stress, inflammation, and heart failure development.
    Article Snippet: .. Mice and Experimental Protocols: Male and female IL-12β knockout (Jackson Laboratory, Strain #002693) and wild-type (WT) C57BL/6J (Jackson Laboratory, Strain #000664) mice were subjected to sham or transverse aortic constriction (TAC) surgery, a procedure used to replicate clinical conditions such as hypertension and aortic stenosis. ..

    Knock-Out:

    Article Title: Genetic inhibition of IL-12β suppresses systolic overload-induced cardiac oxidative stress, inflammation, and heart failure development
    Article Snippet: .. Mice and Experimental Protocols: Male and female IL-12β knockout (Jackson Laboratory, Strain #002693) and wild-type (WT) C57BL/6J (Jackson Laboratory, Strain #000664) mice were subjected to sham or transverse aortic constriction (TAC) surgery, a procedure used to replicate clinical conditions such as hypertension and aortic stenosis. ..

    Article Title: Genetic inhibition of IL-12β suppresses systolic overload-induced cardiac oxidative stress, inflammation, and heart failure development.
    Article Snippet: .. Mice and Experimental Protocols: Male and female IL-12β knockout (Jackson Laboratory, Strain #002693) and wild-type (WT) C57BL/6J (Jackson Laboratory, Strain #000664) mice were subjected to sham or transverse aortic constriction (TAC) surgery, a procedure used to replicate clinical conditions such as hypertension and aortic stenosis. ..



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    Changes in NGF receptors expression upon splenocyte activation: Spleen dissected from wild <t>type</t> <t>C57BL/6J</t> mice were processed to isolate splenocytes and plated as 1 million cells per well in the 96-well plate. After 48hr stimulation either with PMA and iono (PMA:100 ng/ml; iono: 2 μg/ml) or LPS (1 μg/ml), splenocytes were examined for TrkA and p75 by flow cytometry. (A) Histograms present expression of TrkA and p75 in unstimulated (black line), PMA-iono treated (red line) or LPS treated (blue line) T (CD3 + gated) and B (B220 + gated) cells. Unstained controls were displayed as grey dotted line. (B) Bar plot presents percent change in mean fluorescence intensity (MFI) of TrkA and p75 in stimulated groups (PMA-iono or LPS) compared to respective unstimulated (US) group in T (CD3 + gated) and B (B220 + gated) cells. Data are represented as mean ± S.D. and the statistical comparison across different groups was analysed by two-way ANOVA with Tukey’s multiple comparisons test (three independent experiments; n=3 mice); *p < 0.05, ***p < 0.001, ****p < 0.0001.
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    Changes in NGF receptors expression upon splenocyte activation: Spleen dissected from wild type C57BL/6J mice were processed to isolate splenocytes and plated as 1 million cells per well in the 96-well plate. After 48hr stimulation either with PMA and iono (PMA:100 ng/ml; iono: 2 μg/ml) or LPS (1 μg/ml), splenocytes were examined for TrkA and p75 by flow cytometry. (A) Histograms present expression of TrkA and p75 in unstimulated (black line), PMA-iono treated (red line) or LPS treated (blue line) T (CD3 + gated) and B (B220 + gated) cells. Unstained controls were displayed as grey dotted line. (B) Bar plot presents percent change in mean fluorescence intensity (MFI) of TrkA and p75 in stimulated groups (PMA-iono or LPS) compared to respective unstimulated (US) group in T (CD3 + gated) and B (B220 + gated) cells. Data are represented as mean ± S.D. and the statistical comparison across different groups was analysed by two-way ANOVA with Tukey’s multiple comparisons test (three independent experiments; n=3 mice); *p < 0.05, ***p < 0.001, ****p < 0.0001.

    Journal: Frontiers in Immunology

    Article Title: Nerve growth factor responsive elements modulate immune cell inflammation and are dysregulated in an Alzheimer’s disease mouse model

    doi: 10.3389/fimmu.2026.1722477

    Figure Lengend Snippet: Changes in NGF receptors expression upon splenocyte activation: Spleen dissected from wild type C57BL/6J mice were processed to isolate splenocytes and plated as 1 million cells per well in the 96-well plate. After 48hr stimulation either with PMA and iono (PMA:100 ng/ml; iono: 2 μg/ml) or LPS (1 μg/ml), splenocytes were examined for TrkA and p75 by flow cytometry. (A) Histograms present expression of TrkA and p75 in unstimulated (black line), PMA-iono treated (red line) or LPS treated (blue line) T (CD3 + gated) and B (B220 + gated) cells. Unstained controls were displayed as grey dotted line. (B) Bar plot presents percent change in mean fluorescence intensity (MFI) of TrkA and p75 in stimulated groups (PMA-iono or LPS) compared to respective unstimulated (US) group in T (CD3 + gated) and B (B220 + gated) cells. Data are represented as mean ± S.D. and the statistical comparison across different groups was analysed by two-way ANOVA with Tukey’s multiple comparisons test (three independent experiments; n=3 mice); *p < 0.05, ***p < 0.001, ****p < 0.0001.

    Article Snippet: In this study, female wild-type (WT) C57BL/6J (from Janvier labs, France) and App NL-G-F knock-in (NLGF) mice were used.

    Techniques: Expressing, Activation Assay, Flow Cytometry, Fluorescence, Comparison

    Effect of NGF treatment on cytokine production in splenocytes. Spleens dissected from wild type C57BL/6J mice were processed to isolate splenocytes and plated in 96 well plate with 1 million cells per well. Splenocytes were treated with either PMA-iono (PMA:100 ng/ml; iono: 2 μg/ml) or PMA-iono supplemented with 100 ng/ml mature-NGF for 48hr and examined for TNF-α cytokine expression in CD3 + T cells and B220 + B cells by flow cytometry. The bar diagram presents percent change in mean fluorescence intensity (MFI) of TNF-α in stimulated groups (PMA-iono or PMA-Iono + NGF) compared to respective unstimulated (US) group in T (CD3 + ) and B (B220 + ) cells. Data are represented as mean ± S.D. and statistical comparison across different groups (performed as three independent experiments, n=3 mice) were analysed by one-way ANOVA with Tukey’s correction for multiple comparisons; *p < 0.05, **p < 0.01, ***p < 0.001.

    Journal: Frontiers in Immunology

    Article Title: Nerve growth factor responsive elements modulate immune cell inflammation and are dysregulated in an Alzheimer’s disease mouse model

    doi: 10.3389/fimmu.2026.1722477

    Figure Lengend Snippet: Effect of NGF treatment on cytokine production in splenocytes. Spleens dissected from wild type C57BL/6J mice were processed to isolate splenocytes and plated in 96 well plate with 1 million cells per well. Splenocytes were treated with either PMA-iono (PMA:100 ng/ml; iono: 2 μg/ml) or PMA-iono supplemented with 100 ng/ml mature-NGF for 48hr and examined for TNF-α cytokine expression in CD3 + T cells and B220 + B cells by flow cytometry. The bar diagram presents percent change in mean fluorescence intensity (MFI) of TNF-α in stimulated groups (PMA-iono or PMA-Iono + NGF) compared to respective unstimulated (US) group in T (CD3 + ) and B (B220 + ) cells. Data are represented as mean ± S.D. and statistical comparison across different groups (performed as three independent experiments, n=3 mice) were analysed by one-way ANOVA with Tukey’s correction for multiple comparisons; *p < 0.05, **p < 0.01, ***p < 0.001.

    Article Snippet: In this study, female wild-type (WT) C57BL/6J (from Janvier labs, France) and App NL-G-F knock-in (NLGF) mice were used.

    Techniques: Expressing, Flow Cytometry, Fluorescence, Comparison